Preventing the Symptoms of Alzheimer’s Disease

Toxic and radioactive airborne pollutants released during combustion of fuels have been traced to brains where they kill some cells and accelerate the accumulation of amyloid plaques, tau tangles, mercury, lead, and radioactive elements. The accumulations of amyloid plaques and tau tangles associated with Alzheimer’s disease have been found in the brains of children.

Even if you have already accumulated some amyloid plaques and tau tangles in your brain, minimizing your exposure to toxic and radioactive air pollutants may enable you to slow the progression of Alzheimer’s disease and other types of dementia so that none, or only the mildest, of the symptoms, occur during your lifetime.

The safest, most comfortable, and highly energy efficient buildings are tight, highly insulated, and adequately ventilated with fresh filtered air.

Consider using a CO2 monitor and adjusting your ventilation rate to maintain a CO2 level between 600 and 1,000 ppm during the use of heating and cooling systems. Doing so will also help to consistently dilute and flush-out odors, chemicals, microbes, and radon while replenishing oxygen.

After you have a CO2 monitor, and a reliable source of fresh air, consider reducing air leakage through the building envelope to reduce utility bills and minimize entry of air pollutants, insects, drafts, etc.

Air pollution and detrimental effects on children’s brains.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4129915/

Earlier Dementia Onset
https://hurleyeclaw.com/2015/08/07/earlier-dementia-onset/

The “Silent Epidemic of Early-Onset Alzheimer’s
https://thevaccinereaction.org/2017/09/the-silent-epidemic-of-early-onset-alzheimers/

Air Pollution and Children: Neural and Tight Junction Antibodies and Combustion Metals, the Role of Barrier Breakdown and Brain Immunity in Neurodegeneration
https://www.ncbi.nlm.nih.gov/pubmed/25147109

Long-term air pollution exposure is associated with neuroinflammation, an altered innate immune response, disruption of the blood-brain barrier, ultrafine particulate deposition, and accumulation of amyloid beta-42 and alpha-synuclein in the brains of children and young adults.
https://www.ncbi.nlm.nih.gov/pubmed/18349428